Ekg a quick reference guide – aed superstore resource center 6 gases

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Ventricular fibrillation, also called V-fib, occurs when the contraction of the ventricles, normally well coordinated, is disrupted. This occurs when electrical impulses arise from multiple foci within the ventricles causing a chaotic pattern to arise and effectively causing the heart to cease pumping blood [ 1]. electricity sources usa Instead of the ventricles fully contracting to expel blood, V-fib causes the heart to quiver, meaning cardiac output will be non-existent and death will occur rapidly unless life support is put in place.

V-fib is a serious condition that will cause death within a matter of minutes unless properly and promptly treated [ 2]. It is the leading cause of cardiac arrest occurring outside of a hospital setting. V-fib can occur from a number of conditions including MI (Myocardial Infarction), electric shock, severe hypothermia, anti-arrhythmic drugs, and hypoxia among other conditions [ 3]. Defibrillation is the most effective form of treatment for V-fib [ 4]. Some drugs may be administered as well.

Ventricular tachycardia, abbreviated V-tach, is a broad class of arrhythmia consisting of several different types. All forms originate in the lower chambers of the heart. It occurs when 3 or more premature ventricular contractions (PVCs) are present and the rate is over 100 beats per minute [ 5]. The arrhythmia usually occurs due to heart disease, most typically from myocarditis, but also from heart attack, cardiomyopathy, myocardial ischemia etc. It can also occur in younger patients without a history of heart disease and may be difficult to establish cause in such cases [ 6]. V-tach’s various forms and overall unpredictability make it a significant arrhythmia from a clinical perspective.

As previously mentioned ventricular tachycardia may occur in several varieties, with the most common being monomorphic (originating from a single focus) in nature among a handful of other varieties (see Ventricular Tachycardia Monomorphic) [ 7]. The condition may occur in short bursts lasting only briefly and causing brief symptoms or none at all. 6 gases V-tach may also be long lasting in nature and is more serious in this form, requiring immediate treatment to prevent death and serious injury [ 8].

Those exhibiting V-tach very typically show signs of a weak or essentially absent pulse [ 9]. Hypotension will lead to a decreased level of consciousness and, subsequently, will most likely result in a decreased level of responsiveness to outside stimuli [ 10]. Similarly, V-tach may also be caused by changes in blood pressure, lack of oxygen, anti-arrhythmic drugs, or changes in pH [ 11]. Symptoms may include a weak pulse as mentioned, syncope, palpitations, light-headedness, chest pain, and anxiety [ 12]. Tachypnea may also accompany V-tach. gas turbine Treatment is similar to that of V-Fib in cases in which no pulse is detectable [ 13].

Ventricular tachycardia monomorphic (V-tach monomorphic) as opposed to polymorphic V-tach as well as other forms of the arrhythmia such as Torsades de pointes, originates from a single focus located within the ventricle, either left or right [ 14]. As such, all QRS complexes will appear identical, owing to the fact that they are originating from the same place [ 15]. This form of V-tach is especially life threatening when it is sustained which is the most common form of ventricular tachycardia in general.

V-tach monomorphic is likely to impair cardiac output [ 16]. Treatment will thus depend on whether the patient is stable or unstable and is often similar in nature to that of other forms of V-tach [ 17]. gas out game instructions If unstable, treatment will often be in the form of immediate cardioversion. Some patients may only exhibit minor or slight symptoms at first but should still be intervened upon in some form. Due to impaired cardiac output, those exhibiting V-tach very typically show signs of a weak or essentially absent pulse [ 18]. Hypotension most often leads to a decreased level of consciousness and the likely outcome of a subsequent significant decrease in the level of responsiveness to outside stimuli [ 19]. Angina, shortness of breath, and heart failure may also accompany this arrhythmia as well as other forms of V-tach [ 20].

Polymorphic V-tach can occur in brief, even asymptomatic episodes. It can also happen in recurrent, more severe episodes lasting for a longer duration and causing syncope or worse. As such, polymorphic V-tach is a serious condition that can lead to sudden cardiac death and should, therefore, be treated immediately and with care, although the stability of the patient will typically determine treatment [ 23].

Prior heart disease, as well as medication, can cause polymorphic V-tach [ 24]. electricity and circuits Symptoms and treatment of the arrhythmia are usually similar in nature to other forms of V-tach. Some patients, as previously mentioned, may only exhibit minor or slight symptoms initially but should still be intervened upon to some degree. Due to impaired cardiac output, those exhibiting V-tach very typically show signs of a weak or essentially absent pulse [ 25]. Hypotension most often leads to a decreased level of consciousness and the likely outcome of a subsequent significant decrease in the level of responsiveness to outside stimuli [ 26]. Initially, electrolyte imbalances and ischemia are sought to be corrected after which synchronized cardioversion is typically employed [ 27].

Torsade de pointes or TdP is a particular type of polymorphic V-tach frequently occurring in the setting of QT interval prolongation [ 28]. It is characterized by changes in QRS amplitude (morphology will change from beat to beat) and the QRS will appear to twist around the baseline or isoelectric axis on an EKG read although this twisting characteristic may not always be present [ 29]. The arrhythmia is typically not long lasting but rather occurs in brief episodes. la gas prices now However, it re-occurs frequently and is associated with an unstable blood pressure [ 30]. It may change into ventricular tachycardia or ventricular fibrillation.

The treatment of Torsades will generally vary from that of more typical variations of V-tach [ 31]. The cause may be due to drug therapy and is often treated with mechanical override atrial pacing [ 32]. electricity recruitment 2015 Intravenous magnesium is the typical drug used to treat TdP. Symptoms are similar to those of other forms of V-tach including palpitations, dizziness, and syncope. Sudden cardiac death can occur.

In wandering atrial pacemaker arrhythmia the electrical impulses that cause the atrium to contract are wandering or moving from the SA node (the proper site of electrical generation of impulses) to other atrial sites or to the AV junction [ 33]. These changes in impulse origination affect P-wave morphology and thus the appearance of P-waves will typically vary from beat to beat. WAP must exhibit at least three differently shaped P-waves. v gashi halil bytyqi Athletes with well-trained hearts are may demonstrate the arrhythmia, as well as young children. The condition will commonly come and go and thus can be hard to definitively diagnose and many patients do not even realize they have the disorder [ 34].

WAP is most common in older adults and younger children as well as athletes. The arrhythmia may be caused by factors such as increased digoxin toxicity, increased vagal tone, or certain forms of heart disease like rheumatic carditis [ 35]. Typically no treatment is necessary for the arrhythmia and, as stated previously, some patients are not even aware that anything is out of the norm with their hearts or take note when it comes on episodically.

Wolf-Parkinson-White syndrome (WPW for short) occurs when an accessory or alternative heart conduction pathway develops apart from and bypasses travel through the AV junction (the AV junction being the pathway typically responsible for conduction of the electrical signal from the upper atrium to the lower ventricles in a normal functioning heart).These bypass routes of conduction, called bypass tracts, can conduct signals both away from and toward the ventricles (retrograde and anterograde, respectively) and are formed during cardiac development [ 36]. As such, WPW is classified a congenital defect and occurs most frequently in children and those aged 20-35 [ 37]. It is considered a rare disorder and is associated with a slight chance of cardiac death [ 38].